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Smoking-induced insulin resistance, which improves with smoking cessation, may be due to activation of mammalian target of rapamycin (mTOR).

Cigarette smoking increases the risk of subarachnoid hemorrhage (SAH) in a dose-responsive manner, and cessation correlates with a reduction in SAH risk.

For liver transplant recipients, smoking correlates with an increased risk of recurrent viral hepatitis.

Individuals who quit smoking gain a mean of 4-5kg within the first 12 months, with the greatest weight gain occurring during the first three months.

Smokers with certain high-risk genetic variants find it more difficult to quit smoking but are more likely to respond to cessation pharmacotherapy.

Short-term exposure to low levels of secondhand smoke (SHS) results in a concentration-dependent decrease in endothelial function.

Smokers with a new diagnosis of bladder cancer are significantly more likely to quit compared with smokers in the general population.